So what exactly is laminitis and founder? Let’s examine both these conditions to get a better understanding so you can better prevent it from happening.
Laminitis is a crippling disorder of the feet, resulting from damage to the sensitive soft tissues known as ‘laminae’, which connect the skeletal pedal bone to the inside of the hoof wall. There are about 600 interlocking laminae in each hoof and they provide the support for the weight of the horse.
Founder is the physical change that occurs within the hoof as a result of laminitis. When the laminae are damaged, their supporting function is weakened. This may allow the pedal bone within the hoof to effectively ‘sink’ downwards and ‘rotate’ backwards due to the weight of the horse pushing downwards on the bone and the upward pull of the flexor tendon which attaches to the base of the bone. This is an extremely painful condition for the horse, which can become worse as damaged laminae can lead to other conditions such as:
The development of laminitis and founder can often be due to a variety of causes, sometimes acting together and sometimes individually. One of the more common causes however is overeating of feeds rich in ‘soluble carbohydrates’, also referred to as non-structural carbohydrates or NSC (mainly found in cereal grains and lush/stressed pasture).
Metabolic disorders such as EMS (Equine Metabolic Syndrome) and Cushing’s Disease (pituitary pars intermedia dysfunction – PPID) are associated with increased risk of laminitis. In the case of EMS, the underlying issue is a result of hormonal imbalance related to insulin metabolism. Insulin resistance appears to be one of the criteria defining EMS, with clinical signs including obesity-associated laminitis and abnormal fatty deposits or regional adiposity.
PPID on the other hand, is fairly common in older horse (generally over 15 years) and is a disease of the pituitary gland. Clinical signs include laminitis, shaggy coat, undue sweating, lethargy, loss of topline, increased thirst and urination. Gastrointestinal problems such as colic, diarrhoea, retained placenta after foaling, metritis, pneumonia, peritonitis, enteritis etc can also result in laminitis.
Laminitis can also be caused by certain drugs and management factors such as standing for long periods in trucks or floats, poor hoof care and shoeing and by excessive work on hard surfaces (e.g. hoof concussion from galloping or jumping on hard surfaces).
Feeds rich in starch and sugar/fructan can cause a digestive upset in the large intestine. Mammals have no enzyme to digest fructan so this passes undigested directly into the hindgut (caecum and colon). An excess of grain consumption will also ultimately overflow into the hindgut.
In the hindgut, excess starch or fructan will undergo rapid fermentation to lactic acid, with many normal bacteria dying due to the increasingly acidic and low pH conditions. As the acid builds up, it damages the gut linin and toxic substances including bacterial endotoxins and other toxins are released and enter the bloodstream. Once in the bloodstream, the toxins appear to either directly or indirectly stimulate increased production and activity of certain enzymes called matrix metalloproteinases (MMPs). The overactivity of these enzymes destroys the laminae, resulting in the onset of laminitis.
Signs of horse battling grain overload include1:
A common question related to feed-induced laminitis and founder is whether it only affects fat horses and ponies. The resounding answer is definitely NO – all horses can develop laminitis and founder, although pasture-associated laminitis is more common in ponies.
Certain breeds are known to have a higher risk of developing laminitis. Obese horses and ponies are more likely to experience pedal bone rotation and permanent hoof damage due to the extra weight their feet have to support even in a relatively mild case of laminitis.
Surveys have shown that valuable show and equestrian horses are also at risk of developing laminitis even when fed on hard feeds containing relatively small amounts of grain. It is also now known that many racehorses have low-grade laminitis, which may only cause mild sore footedness, a scratchy gait and separation at the ‘white line’. Studies have shown that low-grade laminitis can significantly affect racing performance2.
Yes – once a horse has foundered, it is likely to suffer from continued repeated attacks. These horses can become very sensitive to the carbohydrates in feed, which may trigger a founder episode with only small amounts of grain or lush pasture. Symptoms of laminitis can also occur if the horse’s hooves are not kept correctly trimmed or the animal is worked on hard surfaces.
Laminitis is a distressing and potentially crippling disease which can affect the lifetime performance of your horse. Once a horse develops laminitis and founder, they are more prone to repeat attacks, so prevention is definitely better than a cure in this case. To learn more, read our Laminitis – Prevention is better than cure article.
Products such as FOUNDERGUARD contain ‘Virginiamycin’ and can be added to the feed to help prevent laminitis from occurring. It works by suppressing the activity of the ‘bad’ bacteria which produces lactic acid, thus maintaining the gut microbes in the correct balance. FOUNDERGUARD is a preventative rather than a treatment and cannot correct any physical damage that has already occurred in the feet. However, following an attack of laminitis, FOUNDERGUARD can help prevent the ‘flare-ups’ that frequently occur in the recovery period.
FOUNDERGUARD can also be added to the horse’s feed to prevent low-grade laminitis, which is a mild form of laminitis with no outward signs, so the disease often goes unnoticed. If you suspect your horse has a case of laminitis, call your veterinarian for advice. The sooner you call, the more likely you will be able to successfully treat your horse’s laminitis and limit any lifelong effects. Prevention really is better than cure.
Reduce the risk of laminitis in your horse with FOUNDERGUARD®